I think this whole plastic-issue is vastly overblown. For one thing, you have to have these liquids stored in plastic for years before the chemicals start leaching into the liquids. Also, if one doesn't store the bottles in open sunlight, the process takes longer.
I've been drinking excellent alkaline mineral-water from natural springs enclosed in plastic and have never had an issue.
Well, I agree with Hannibal. At least during pregnancy and crucial steps of fetal development this kind of chemicals that mimic estrogens is a
very serious issue and a well documented threat as shown by the following paper among many others:
Environmental Research 100 (2006) 39–43
"Increased serum estrogenic bioactivity in three male newborns with ambiguous genitalia: A potential consequence of prenatal exposure to environmental endocrine disruptors."
Francoise Paris a,b,c, Claire Jeandela, Nadege Servant b,c, Charles Sultan a,b,c,a
Unite ? d’Endocrinologie Pe ?diatrique, Service de Pe ?diatrie 1, Hoˆpital Arnaud de Villeneuve, CHU Montpellier, 34295 Montpellier Cedex 5, France bINSERM Unite ? 540 (Groupe Pathologie Mole ?culaire des Androge`nes), Montpellier, France cService d’Hormonologie du De ?veloppement et de la Reproduction, CHU Montpellier, Montpellier, France Received 8 December 2004; received in revised form 3 June 2005; accepted 22 June 2005 Available online 7 November 2005
Abstract
In the past 15 years, anomalies of male sexual differentiation have greatly increased in both wildlife and humans in different parts of the world. Environmental endocrine disruptors have been implicated in the dramatic rise in neonatal ambiguous genitalia with variable rates of severity, such as micropenis, cryptorchidism, and isolated or associated hypospadias. Because most environmental pollutants, such as organochlorine pesticides, polychlorinated biphenyls, dioxins and furans, alkylphenol polyetholyethoxylates, and phytoestrogens and phtalates, have estrogenic and antiandrogenic activity, they are able to interfere with normal fetal male sexual differentiation. In a neonatal screening program of ambiguous genitalia, we had the opportunity to evaluate three newborns with male pseudohermaphroditism (MPH) whose mothers were exposed to endocrine disruptors during pregnancy. All had normal testosterone production after human chorionic gonadotrophin stimulation testing, suggesting androgen resistance or so-called idiopathic MPH. Sequences of the 5a reductase and androgen receptor genes were normal. Since environmental pollutants are known for their estrogenic activity and can be released progressively from the adipose tissue where they accumulate, we detected their presence by measuring the estrogenic bioactivity of the newborns’ serum with a recently developed ultrasensitive bioassay. We found higher estrogenic bioactivity in these newborns than in controls. In conclusion, the maternal exposure to environmental pollutants during pregnancy and high estrogenic bioactivity in the newborns’ serum highly suggest that ambiguous genitalia are related to fetal exposure to endocrine disruptors.