I was just doing my usual reading on this website -
www.microbialinfluence.com - it seems every time I look I find something new.
http://www.microbialinfluence.com/NFKB.html"NF-?B (nuclear factor-kappa B) is a protein complex that plays a key role in
regulating the immune response to infection. Unfortunately, incorrect regulation of NF-?B has been linked to cancer, inflammatory and autoimmune diseases, septic shock,
viral infection, and improper immune development."
Maybe there's something to what the microbes in the gut are doing and herpes.
I know when I saw this, I was shocked, and I'm sure the same applies to herpes.
"Lipopolysaccharide (LPS) potently stimulates human immunodeficiency virus type 1-long terminal repeat (HIV-1-LTR) CAT constructs transfected into monocyte/macrophage-like cell lines but not a T cell line. This effect appears to be mediated through the induction of nuclear factor kappa B (NF-kappa B). Electrophoretic mobility shift assays demonstrate that LPS induces a DNA binding activity indistinguishable from NF-kappa B in U937 and THP-1 cells. LPS is also shown to dramatically increase HIV-1 production from a chronically infected monocyte/macrophage-like cloned cell line, U1, which produces very low levels of HIV-1 at baseline. The stimulation of viral production from this cell line occurs only if these cells are treated with granulocyte/macrophage colony-stimulating factor (GM-CSF) before treatment with LPS. This stimulation of HIV-1 production is correlated with an increase in the level of HIV-1 RNA and and activation of NF-kappa B. LPS is not able to induce HIV-1 production in a cloned T cell line. The effect of LPS on HIV-1 replication occurs at picogram per milliliter concentrations and may be clinically significant in understanding the variability of the natural history of HIV-1 infection."
If something that goes on with the microbes can affect the rate HIV replicates, who knows if tweeking the microbes can give you resistance to HIV, herpes, etc. Like Thoth has stated, he is symptom free even after being exposed to various typically pathogenic bacteria/virus.